Nov, 2022 - By SMI
Blood’s cellular compositions seems to alter due to COVID-19 infection; this serves as a leading step in research to study the cellular difference of with and without COVID-19 infected diabetic patient.
Dr. Dinender Singla. Specialist and College of Medicine researcher in diabetes, inflammation and heart failure, had recently published an article in the American Journal of Physiology- Heart and Circulatory Physiology. The article reads the examination done on the mechanisms of COVID- 19 risk involved with diabetes and the advancement of virus leading to inflammation and heart failure.
The genotype of diabetic patients or pre diabetic patients grow more prone to inflammatory conditions after COVID that affects brain and heart.
Dr. Singla marked that COVID-9 plays long term effects potentially on patients that fall under three categories as, patients of Cognitive Dysfunction can adverse to Alzheimer’s disease, the risk of diabetes increases for patients with pre-diabetic conditions and the worsening of existing diabetic patients to complications such as muscle dysfunction and cardiomyopathy.
The study is leading in the way to study the cellular difference of diabetic patients with and without COVID infection. Cytokines, the protein involved in communication among cells is looked for any changes after COVID infection. Any difference in protein needs to be examined for diseases it could develop.
Cardiovascular disease, along with diabetes and hypertension, increases the risk of infection and results in poor outcomes. COVID-19 virus SARS-CoV-2 penetrates host cells via angiotensin-converting enzyme-2 receptor, causing inflammation and cytokine uproar that frequently leads to multiorgan failure. The overall mechanisms and effects of COVID-19 on patients with high-risk diabetes are still to fully understood.
The researchers are looking forward to study in depth COVID-19 infection in long term i.e., 10 or 20 years from today, the response of virus to vaccines and advancement of virus with other existing diseases.
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